Reasons why insulin resistance can lead to belly obesity.

Insulin resistance can cause abdominal obesity through physiological causes. Pancreatic insulin regulates blood sugar (glucose) levels and helps cells store excess glucose. 

 When cells become insulin-resistant, glucose absorption is less effective, raising blood sugar. Insulin resistance may cause belly fat for several reasons:

High Insulin Levels: To overcome insulin resistance and control blood sugar, the body initially produces more insulin. However, high insulin levels can enhance belly fat storage.

Insulin helps cells store glucose and lipids. When cells become insulin-resistant, muscle cells cannot retain glucose and more glucose goes to fat cells. This increases fat storage, especially abdominal visceral fat.

Insulin resistance increases lipogenesis, the process by which the body turns carbs into fat. This can increase belly fat.

Impaired Lipolysis: Insulin normally suppresses fat breakdown. Insulin resistance reduces this inhibition, releasing more fatty acids from fat cells into the bloodstream. 

Chronic low-grade inflammation is linked to insulin resistance. Inflammation affects visceral fat formation. Metabolically active visceral fat releases inflammatory chemicals that worsen insulin resistance, generating a feedback loop.

Insulin resistance can be treated by eating well, exercising, and maintaining a healthy weight. If you're worried about insulin resistance or abdominal obesity, see a doctor for a personalized assessment and treatment.

Watch this space for further developments.